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Shabbir W, Topcagic N, Aufy M, Oz M. CRISPR/Cas9 Mediated Knock Down of δ-ENaC Blunted the TNF-Induced Activation of ENaC in A549 Cells. International Journal of Molecular Sciences. 2021 Feb 2;22(4):1-10. 1858. doi: 10.3390/ijms22041858
Iqbal SM, Aufy M, Shabbir W, Lemmens-Gruber R. Identification of phosphorylation sites and binding pockets for modulation of NaV 1.5 channel by Fyn tyrosine kinase. The FEBS Journal. 2018 Jul;285(13):2520-2530. doi: 10.1111/febs.14496
Willam A, Aufy M, Tzotzos S, El-Malazi D, Poser F, Wagner A et al. TNF Lectin-Like Domain Restores Epithelial Sodium Channel Function in Frameshift Mutants Associated with Pseudohypoaldosteronism Type 1B. Frontiers in Immunology. 2017 Mai 29;8:601. doi: 10.3389/fimmu.2017.00601
Willam A, Aufy M, Tzotzos S, Evanzin H, Chytracek S, Geppert S et al. Restoration of Epithelial Sodium Channel Function by Synthetic Peptides in Pseudohypoaldosteronism Type 1B Mutants. Frontiers in Pharmacology. 2017 Feb 24;8:85. doi: 10.3389/fphar.2017.00085
Lucas R, Yue Q, Alli A, Duke BJ, Al-Khalili O, Thai TL et al. The Lectin-like Domain of TNF Increases ENaC Open Probability through a Novel Site at the Interface between the Second Transmembrane and C-terminal Domains of the α-Subunit. Journal of Biological Chemistry. 2016 Nov 4;291(45):23440-23451. doi: 10.1074/jbc.M116.718163
Shabbir W, Tzotzos S, Bedak M, Aufy M, Willam A, Kraihammer M et al. Glycosylation-dependent activation of epithelial sodium channel by solnatide. Biochemical Pharmacology. 2015 Dez 15;98(4):740-753. doi: 10.1016/j.bcp.2015.08.003
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